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KeptByTheWord
04-22-2020, 12:33 AM
Excerpt from the article:
Now, this team of scientists has discovered 31 new mutated strains of the virus that might explain the stubbornly high mortality rates in parts of Europe and New York.

According to the South China Morning Post, some of the mutant strains exhibited a much more dangerous capacity to invade human cells, implying that certain strains might be much more lethal than others. What's more, these strains were found to be "genetically similar" to samples isolated in New York and places like Italy in Europe.

and another excerpt:

The fact that such unexpectedly intense variations could arise from a sample of fewer than a dozen patients means the genetic variability of this virus might be much higher than initially expected. And it may have mutated since the outbreak began, which of course could create complications in the quest for a vaccine. Most alarmingly, some of the mutated strains carried as much as 270x the viral load as the weakest strains.

This is a very interesting study. This would explain "hot spots" and why the virus seems to kill more people in some areas, while other areas are mostly asymptomatic.

I'm also encouraged to see that the use of vaccination in this type of scenario is extremely unlikely.

diakonos
04-22-2020, 05:10 AM
According to the South China Morning Post... 🥢🥡

james34
04-22-2020, 06:25 AM
Excerpt from the article:
Now, this team of scientists has discovered 31 new mutated strains of the virus that might explain the stubbornly high mortality rates in parts of Europe and New York.

According to the South China Morning Post, some of the mutant strains exhibited a much more dangerous capacity to invade human cells, implying that certain strains might be much more lethal than others. What's more, these strains were found to be "genetically similar" to samples isolated in New York and places like Italy in Europe.

and another excerpt:

The fact that such unexpectedly intense variations could arise from a sample of fewer than a dozen patients means the genetic variability of this virus might be much higher than initially expected. And it may have mutated since the outbreak began, which of course could create complications in the quest for a vaccine. Most alarmingly, some of the mutated strains carried as much as 270x the viral load as the weakest strains.

This is a very interesting study. This would explain "hot spots" and why the virus seems to kill more people in some areas, while other areas are mostly asymptomatic.

I'm also encouraged to see that the use of vaccination in this type of scenario is extremely unlikely.
The US has to much vaccination momentum to back out now. We are going down that road.

coksiw
04-22-2020, 10:44 AM
Coronaviruses are very similar to each other, to the point that immunity to some of them can help with the other strains. That's the reason why children don't have such a high mortality, or even critical symptoms. It is estimated that children get a coronavirus (common cold) every month.

The other thing is that if the virus gets bigger with more load in it, usually means that their transmissibility lowers as well.

Esaias
04-23-2020, 04:03 AM
Wait till you get red pilled on viruses vs exosomes...

KeptByTheWord
04-24-2020, 12:53 AM
Or how about HIV virus mixed with Coronavirus?

https://www.zerohedge.com/health/renowned-microbiologist-claims-wuhan-lab-did-absolutely-crazy-things-coronavirus

Excerpt: "A world renowned Russian microbiologist says that the novel coronavirus responsible for the COVID-19 pandemic was the result of Wuhan scientists doing "absolutely crazy things" in their laboratory."

Excerpt: "Chumakov suggested that Chinese scientists were possibly searching for an HIV vaccine."

Then read this article on a recent finding that HIV attacks cells, much like HIV:

https://www.thailandmedical.news/news/covid-19-alert-new-study-shows-sars-cov-2-coronavirus-targets-and-destroys-t-cells,-similarly-as-what-hiv-does

Excerpt: "COVID-19 Alert: To add to many studies already showing that the deadly and potent ways the SARS-Cov-2 has of attacking various human host cells and also critical proteins and cellular pathways in the human cells, a new collaborative study by researchers from the United States and China now shows that the SARS-CoV-2 coronavirus also attacks the T Lymphocytes similarly to the way that HIV viruses act."

Esaias
04-24-2020, 04:16 AM
Or how about HIV virus mixed with Coronavirus?

https://www.zerohedge.com/health/renowned-microbiologist-claims-wuhan-lab-did-absolutely-crazy-things-coronavirus

Excerpt: "A world renowned Russian microbiologist says that the novel coronavirus responsible for the COVID-19 pandemic was the result of Wuhan scientists doing "absolutely crazy things" in their laboratory."

Excerpt: "Chumakov suggested that Chinese scientists were possibly searching for an HIV vaccine."

Then read this article on a recent finding that HIV attacks cells, much like HIV:

https://www.thailandmedical.news/news/covid-19-alert-new-study-shows-sars-cov-2-coronavirus-targets-and-destroys-t-cells,-similarly-as-what-hiv-does

Excerpt: "COVID-19 Alert: To add to many studies already showing that the deadly and potent ways the SARS-Cov-2 has of attacking various human host cells and also critical proteins and cellular pathways in the human cells, a new collaborative study by researchers from the United States and China now shows that the SARS-CoV-2 coronavirus also attacks the T Lymphocytes similarly to the way that HIV viruses act."

You must have missed this: http://www.apostolicfriendsforum.com/showthread.php?t=53726

https://www.biorxiv.org/content/10.1101/2020.01.30.927871v1

Abstract

We are currently witnessing a major epidemic caused by the 2019 novel coronavirus (2019- nCoV). The evolution of 2019-nCoV remains elusive. We found 4 insertions in the spike glycoprotein (S) which are unique to the 2019-nCoV and are not present in other coronaviruses. Importantly, amino acid residues in all the 4 inserts have identity or similarity to those in the HIV-1 gp120 or HIV-1 Gag. Interestingly, despite the inserts being discontinuous on the primary amino acid sequence, 3D-modelling of the 2019-nCoV suggests that they converge to constitute the receptor binding site. The finding of 4 unique inserts in the 2019-nCoV, all of which have identity /similarity to amino acid residues in key structural proteins of HIV-1 is unlikely to be fortuitous in nature. This work provides yet unknown insights on 2019-nCoV and sheds light on the evolution and pathogenicity of this virus with important implications for diagnosis of this virus.

That was 2 February.

Now... What if I told you HIV doesn't cause AIDS? And then what if I told you there's no such thing as Human Immunodeficiency Virus?

And then what if you found out that Dr Fauci and Dr Birx were part of the creation of the HIV/AIDS scandemic way back in the 80s?